Gingival Inflammation Question and Answers

Gingival Inflammation Important Notes

1. Stages of gingivitis

Gingival Inflammation Stages of gingivitis

2. Stage 3 gingivitis

Clinical features:

  • Engorgement of blood vessels
  • Bluish red appearance of the gingiva
  • Changes in size and texture

Histological features:

  • Infiltration of connective tissue and junctional epithelium with neutrophils, and lymphocytes with the predominance of plasma cells

3. Gingival pigmentation caused by heavy metals

Gingival Inflammation Gingival pigmentation caused by heavy metals

Gingival Inflammation Short Essays

Question 1. Stages of Gingivitis.
Answer:

Stage 1 Initial Lesion:

  • Classic vasculitis of vessels
  • Exudation of fluid from gingival sulcus
  • Changes in the coronal portion of JE
  • Migration of leukocytes
  • Presence of serum proteins
  • Loss of collagen

Gingival Inflammation Initial lesion

Stage 2-Early Lesion:

  • Erythematous Gingiva
  • Bleeding on probing
  • Development of recipes in JE
  • Presence of lymphocytes
  • Cytotoxic changes of fibroblasts
  • Loss of collagen

Stage 3-Established Lesion:

  • Bluish hue on reddened gingiva
  • Moderate to severely inflamed gingiva
  • Presence of plasma cells deep into connective tissue

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  • Apical migration of junctional epithelium
  • Collagen destruction
  • Continuous loss of ground substance
  • Pocket formation
  • Elevated levels of acid and alkaline phosphate, B- glucuronidase, aminopeptidase

Stage 4-Advanced Lesion:

  • Persistence of infection
  • Bone loss
  • Loss of collagen
  • Presence of all inflammatory cells Formation of pockets
  • Leads to periodontics

Gingival Inflammation Early lesion

Question 2. Gingival pigmentation.
Answer:

  • The color of gingiva is determined by several factors in which pigments within the epithelium are one of the factors

Types:

  1. Endogenous pigmentation
  2. Exogenous pigmentation

Gingival Inflammation Endogenous pigmentation and Exogenous pigmentation

Causes:

1. Localized:

  • Amalgam tattoo
  • Graphite
  • Nevus
  • Melanotic macules
  • Malignant melanoma
  • Kaposi’s sarcoma

2. Generalized pigmentation:

  • Genetics- Peutz-Jegher’s syndrome
  • Habits- Smoking, betel chewing
  • Drugs
    • Antimalarial
    • Antimicrobial
    • Minocycline
    • Ketoconazole
    • Contraceptive pills
    • Heavy metal exposure
  • Endocrine
    • Addison’s disease
    • Albright’s syndrome
    • Pregnancy
  • Post-inflammatory
    • Periodontal disease
    • Post-surgical gingival pigmentation
  • Others
    • Haemochromatosis
    • Generalised neurofibromatosis
    • HIV disease
    • Thalassaemia

Question 3. Stage 2-Early lesion.
Answer:

Stage 2-Early Lesion:

  • Erythematous Gingiva
  • Bleeding on probing
  • Development of rete pegs in JE
  • Presence of lymphocytes
  • Cytotoxic changes of fibroblasts
  • Loss of collagen

Gingival Inflammation Short Answers

Question 1. Stage III of gingivitis.
Answer:

  • Stage 3 gingivitis is an established lesion

Features:

  • Bluish hue on reddened gingiva
  • Moderate to severely inflamed gingiva
  • Presence of plasma cells deep into connective tissue
  • Apical migration of junctional epithelium
  • Collagen destruction
  • Continuous loss of ground substance
  • Pocket formation
  • Elevated levels of acid and alkaline phosphate, beta-glucuronidase, aminopeptidase

Q. 2. Plasma cell gingivitis.
Answer:

  • Plasma cell gingivitis is also referred to as atypical and plasma cell gingivostomatitis
  • It consists of a mild marginal gingival enlargement that extends to the attached gingiva
  • Gingiva appears red, friable, and bleeds easily
  • Microscopically, connective tissue contains a dense infiltrate of plasma cells that also extends to oral epithelium

Auses:

  • Allergy
  • Related to components of chewing gums or denitrifies

Gingival Inflammation Viva Voce

  1. The predominant cell in acute gingivitis is the T lymphocyte
  2. Gingivitis is the most common form of gingival disease
  3. Bacteria found in gingivitis are localized in the gingival sulcus
  4. Extension of inflammation into the supporting structures occurs in stage 4
  5. Stage I gingivitis is subclinical
  6. Erythema and bleeding on probing occurs in stage 2
  7. The established lesion of gingivitis is characterized by the predominance of plasma cells and B lymphocytes
  8. The B cells found in established lesions are predominantly IgG and IgG, subclasses

 

 

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